Impairments in Site-Specific AS160 Phosphorylation and Effects of Exercise Training
نویسندگان
چکیده
منابع مشابه
Impairments in Site-Specific AS160 Phosphorylation and Effects of Exercise Training
The purpose of this study was to determine if site-specific phosphorylation at the level of Akt substrate of 160 kDa (AS160) is altered in skeletal muscle from sedentary humans across a wide range of the adult life span (18-84 years of age) and if endurance- and/or strength-oriented exercise training could rescue decrements in insulin action and skeletal muscle AS160 phosphorylation. A euglycem...
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15 صفحه اولExercise-induced phosphorylation of the novel Akt substrates AS160 and filamin A in human skeletal muscle.
Skeletal muscle contraction stimulates multiple signaling cascades that govern a variety of metabolic and transcriptional events. Akt/protein kinase B regulates metabolism and growth/muscle hypertrophy, but contraction effects on this target and its substrates are varied and may depend on the mode of the contractile stimulus. Accordingly, we determined the effects of endurance or resistance exe...
متن کاملPrior exercise increases phosphorylation of Akt substrate of 160 kDa (AS160) in rat skeletal muscle.
The main purpose of this study was to determine whether the increased glucose transport (GT) found immediately postexercise (IPEX) or 4 h postexercise (4hPEX) is accompanied by increased phosphorylation of Akt substrate of 160 kDa (AS160, a protein regulator of GLUT4 translocation). Paired epitrochlearis muscles were dissected from rats (sedentary or IPEX, 2-h swim) and used to measure protein ...
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Cognitive impairments describe a state of diminished or impaired mental and/or intellectual function such as Alzheimer's disease, Huntington's disease and Parkinson's disease. As these disorders are more frequent in the elderly and due to the ageing of population, serious attention must be paid to these diseases. Exercise has shown to have preventive and therapeutic effects on cognitive impairm...
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ژورنال
عنوان ژورنال: Diabetes
سال: 2013
ISSN: 0012-1797,1939-327X
DOI: 10.2337/db13-0229